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By Reinhard L. Friede

I used to be gratified by means of the main favorable reception and extensive utilization got by way of the 1st version of this publication. A decade seems a brief interval for a booklet on pathology, and but it witnessed many vital alterations of techniques, besides an impressive development of information. the second one variation required vast reorganization. There are new chapters on mitochondriopathies, on peroxisomal ailments and on spongy myelino­ pathies. significant revisions and new additions have been worthy in lots of chapters, for example these at the dysplasias of the cerebral and of the cerebellar hemispheres, which have been mostly reorganized. The chapters on perinatal pathology have been reordered and reorganized to provide a extra logical series of prenatal, perinatal and postnatal lesions. the full textual content was once labored over for brevity. A wealth of recent references was once further with the. target of staying abreast with the literature as much as summer season 1988. All refer­ ences have been double checked for blunders. My gratitude is going to Mrs. Gisela Ropte and Mrs. Cynthia Bunker for his or her untiring, diligent aid. accordingly, this moment version is an primarily rewritten textual content. strengthen within the prevention of human agony is predicated on a radical comprehend­ ing of the character of disorder. i am hoping that this article is going to remain of carrier during this behalf. probably it will possibly additionally replicate and foster the highbrow interest which makes the "reading of brains" so fascinating an profession. Gottingen, 1989 Reinhard L.

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Developmental Neuropathology

I used to be gratified by way of the main favorable reception and large utilization got by way of the 1st variation of this booklet. A decade seems a quick interval for a booklet on pathology, and but it witnessed many vital alterations of strategies, in addition to a powerful development of data. the second one version required vast reorganization.

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Anatomical study. BioI. Neonat. 20: 287-299, 1972. - Sub-ependymal pseudocysts in the newborn. BioI. Neonat. 21: 170-183,1972. Leech, R. : Subependymal and intraventricular hemorrhages in the newborn. Am. ]. Path. 77: 465-476, 1974. : The aetiology of neonatal hydrocephalus. Develop. Med. Child Neurol. 7: 289-294, 1965. Miller, J. : Prevention of brain damage during asphyxia. Proc. 2nd Congo Int. Asso. Sci. Study Mental Deficiency, Poland, 1970, pp. 444-449. Naeye, R. , Wright, D. : Neonatal mortality, the male disadvantage.

70: 137-151, 1939. Lucas Keene, M. , Hewer, E. : Some observations on myelination in the human central Anat. 66: 1-13, 1931. nervous system . McFarland, D. , Friede, R. : Number of fibers per sheath cell and internodal length in cat cranial nerves. J Anat. 109: 169-176, 1971. Martinez, A. , Friede, R. : Changes in nerve cell bodies during the myelination of their axons. J compo Neurol. 138: 329-338, 1970. Matthews, M. : A quantitative study of morphological changes accompanying the initiation and progress of myelin production in the dorsal funiculus of the rat spinal cord .

Towbin's (1968) claim that phlebothrombosis with resultant matrix infarction is generally responsible for intraventricular hemorrhages lacks convincing histologic documentation. Definite fibrinous thrombi in dilated veins were documented in isolated instances by Larroche (1954) and Ross and Dimmette, 2 of 30 cases, (1965) and were observed occasionally in the present author's material. These cases, however, consistute only a small fraction of all intraventricular hemorrhages; the thrombi may develop in reaction to the hemorrhage (Claireaux, 1959), or upon prolonged stasis in the congested vessels.

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