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1992). , 1994), have been developed. In conclusion, since 1980, the rapid development of new methods has greatly increased our understanding of airway epithelial function. The profound impact of this tissue on the function of surrounding cells and tissues has become appreciated. Finally, detailed information is now available on how malfunction of airway epithelium contributes to the pathology of CF, asthma, and chronic bronchitis. K. (1988) Proliferation and differentiation in mammalian airway epithelium.

Pathology . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 20 20 23 24 32 Submucosal Glands . . . Structure and Development Function . . . . . . Pathology . . . . . Summary . . . . . . . . . . . . . . . . . . . . . , 1991). , 1995) has revealed many new functions. It is now realized, for instance, that airway epithelia are capable of affecting rapid changes in the depth and composition of the thin film of liquid that lines the airways.

Role of glands in mucus accumulation in cystic fibrosis. In a normal gland, chloride secretion by serous cells in the acinus mediates the secretion of isotonic salt solution that flushes out mucins from the gland tubules. In cystic fibrosis, failure of acini to secrete Cl and water leads to a progressive build up of concentrated mucous secretions that distend the gland lumen. The dehydrated mucous secretions may fail to detach from the gland opening or may be inefficiently transported by the cilia, gradually accumulating on the airway surface.

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